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ANP (Atrial Natriuretic Peptide) vs NA-Selank Amidate

A detailed comparison to help you understand the differences and choose the right peptide for your research goals.

ANP (Atrial Natriuretic Peptide)

ANP is a cardiac hormone released by atrial myocytes in response to stretch. It promotes natriuresis, diuresis, and vasodilation, playing key roles in blood pressure and fluid regulation.

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NA-Selank Amidate

NA-Selank Amidate (N-Acetyl Selank Amidate) is an enhanced version of Selank with improved stability and blood-brain barrier penetration. The modifications increase bioavailability and duration of cognitive and anxiolytic effects.

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Side-by-Side Comparison

AspectANP (Atrial Natriuretic Peptide)NA-Selank Amidate
MechanismBinds to natriuretic peptide receptors (NPR-A) to activate guanylyl cyclase, producing cGMP. This leads to vasodilation, increased kidney filtration, and inhibition of the renin-angiotensin-aldosterone system.Same core mechanism as Selank - modulates BDNF, serotonin, and norepinephrine systems. The N-acetyl group improves membrane permeability while the amidate modification increases enzymatic stability.
Typical DosageClinical use: Carperitide (recombinant ANP) used in Japan for acute heart failure at 0.1mcg/kg/min IV infusion.Intranasal: 100-400mcg 1-3 times daily. Lower doses needed compared to standard Selank due to enhanced bioavailability.
AdministrationIntravenous infusion only for clinical applications. Short half-life (~2 minutes) requires continuous administration.Primarily intranasal administration. Can be used sublingually. More stable in solution than standard Selank.
Side EffectsHypotension (dose-limiting), headache, nausea, and potential arrhythmias at high doses.Generally well-tolerated. Possible mild fatigue or nasal irritation. Less frequent dosing needed than standard Selank.
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Key Differences

Unique to ANP (Atrial Natriuretic Peptide):

Unique to NA-Selank Amidate:

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