Thymulin vs ANP (Atrial Natriuretic Peptide)

A detailed comparison to help you understand the differences and choose the right peptide for your research goals.

Thymulin

Thymulin (Facteur Thymique Sérique) is a zinc-dependent nonapeptide hormone produced by thymic epithelial cells. It plays important roles in T-cell differentiation and immune system maturation.

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ANP (Atrial Natriuretic Peptide)

ANP is a cardiac hormone released by atrial myocytes in response to stretch. It promotes natriuresis, diuresis, and vasodilation, playing key roles in blood pressure and fluid regulation.

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Side-by-Side Comparison

AspectThymulinANP (Atrial Natriuretic Peptide)
MechanismRequires zinc for biological activity. Promotes T-cell differentiation, modulates cytokine production, and influences neuroendocrine function. Levels decline significantly with age.Binds to natriuretic peptide receptors (NPR-A) to activate guanylyl cyclase, producing cGMP. This leads to vasodilation, increased kidney filtration, and inhibition of the renin-angiotensin-aldosterone system.
Typical DosageResearch protocols vary. Often studied alongside zinc supplementation. Typical research doses in the microgram range.Clinical use: Carperitide (recombinant ANP) used in Japan for acute heart failure at 0.1mcg/kg/min IV infusion.
AdministrationSubcutaneous or intramuscular injection in research settings. Requires adequate zinc status for activity.Intravenous infusion only for clinical applications. Short half-life (~2 minutes) requires continuous administration.
Side EffectsLimited data on exogenous administration. Theoretical effects on immune function require monitoring.Hypotension (dose-limiting), headache, nausea, and potential arrhythmias at high doses.
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Key Differences

Unique to Thymulin:

Unique to ANP (Atrial Natriuretic Peptide):

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