Kisspeptin-10 vs Palmitoyl Tripeptide-1

A detailed comparison to help you understand the differences and choose the right peptide for your research goals.

Kisspeptin-10

Kisspeptin-10 is the active fragment of kisspeptin, a hormone that plays a crucial role in initiating puberty and regulating reproductive function. It acts upstream of GnRH in the hypothalamic-pituitary-gonadal axis.

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Palmitoyl Tripeptide-1

Palmitoyl Tripeptide-1 (Pal-GHK) is a lipopeptide that stimulates collagen production. It's one of two peptides in the Matrixyl 3000 complex, working synergistically with Palmitoyl Tetrapeptide-7.

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Side-by-Side Comparison

AspectKisspeptin-10Palmitoyl Tripeptide-1
MechanismBinds to KISS1R receptors in the hypothalamus, stimulating GnRH neurons to release GnRH. This triggers the downstream cascade of LH, FSH, and sex hormone production.Mimics the skin's own mechanism for producing collagen by acting as a messenger peptide that signals fibroblasts to produce more collagen and other extracellular matrix components.
Typical DosageResearch protocols vary widely. Studies have used IV boluses of 1-10mcg/kg or subcutaneous administration. Clinical applications still being developed.Topical: Typically 2-4% in serums, often combined with Palmitoyl Tetrapeptide-7 as Matrixyl 3000.
AdministrationIV or subcutaneous injection. Has very short half-life requiring frequent administration or continuous infusion for sustained effects.Topical application 1-2 times daily. The palmitoyl group enhances skin penetration compared to non-lipidated versions.
Side EffectsLimited data. May cause flushing, increased heart rate, and changes in libido. Generally well-tolerated in studies.Very well-tolerated. Suitable for most skin types including sensitive skin.
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Key Differences

Unique to Kisspeptin-10:

Unique to Palmitoyl Tripeptide-1:

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